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anonymous

  • one year ago

This is Microbiology, but couldn't find the subject. Vancomycin-resistant Staphylococcus aureus (vrsa) was isolated from the exit site of a dialysis catheter in a 40-year-old diabetic with a history of periphreal vascular disease, chronic renal failure, and chronic foot ulcers. A few months earlier, the patient's gangrenous toe had been amputated. Following that surgery, the patient developed bacteremia. Vancomycin, rifampin, and graft removal successfully treated to infection. A few months later, when the catheter exit site infection appeared, the are was cultured & the catheter removed,

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  1. anonymous
    • one year ago
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    Hello welcome to open study!

  2. anonymous
    • one year ago
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    i dont think she is new

  3. anonymous
    • one year ago
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    successfully treating the infection. A week later the patient's chronic foot ulcer again appeared infected. Vancomycin-resistant Enterococcus faecalis (VRE) and Klebsiella oxytoca were cultured from the ulcer. The patient recovered after wound care and systemic treatment with trimethoprim/sulfamethoxozale.

  4. anonymous
    • one year ago
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    Analysis of the VRSA isolate revealed that it contained the van A gene for vancomycin resistance and the mec A gene for oxacillin resistance. -How do you think the Staphylococcus aureus strain ended up with the gene for vancomycin resistance? -What is one possible mechanism for genetic transfer of antibiotic resistance from one organism to another? -Why would this particular patient be at increased rick for infection with VRSA? -Provide a detailed explanation of how genes, such as those responsible for drug resistance, be transferred between bacterial species? -Where could S. aureus likely have acquired the genes for drug resistance? and -What do we call infections that are acquired in a medical setting AND how are they tracked within hospital facilities?

  5. anonymous
    • one year ago
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    What do you think?

  6. anonymous
    • one year ago
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    I think for the first question the S.aureus strain ended up with the gene for vancomycin when the patient developed the bacreremia. The second one I'm at a loss for. The third one I think the patient would be at an increased risk because of the resistances (is that even a word? lol) the patient has. The fourth one I'm also at a loss for. The fifth one, isn't that like the first question? And i'm not sure about the last one.

  7. nincompoop
    • one year ago
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    you are on the right track. gene transfer occur from one bacteria to another and then self-mutation may have happened. Usually, complete treatment does not involve 100% success and may have still missed a few bacteria that have mutated during its growth and produced characteristics already resistant to the treatment.

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